October 2019 — Vol 139 No 10

GRaphical Abstract showing that  AD-like inflammation in skin isdependent on basophils and ILC2s. Basophil-derived IL-4 may be a keymediator.

In a transgenic mouse model in which IL-33 is constitutively overproduced bykeratinocytes, Imai and colleagues show that AD-like inflammation in skin isdependent on basophils and ILC2s. Basophil-derived IL-4 may be a keymediator. Please see the article on page 2185 and the relevant commentaryon page 2077 of this issue for details.

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