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    • Original Article Wound Healing
      Open Archive

      Caveolin-1 Controls Hyperresponsiveness to Mechanical Stimuli and Fibrogenesis-Associated RUNX2 Activation in Keloid Fibroblasts

      Journal of Investigative Dermatology
      Vol. 138Issue 1p208–218Published online: September 9, 2017
      • Chao-Kai Hsu
      • Hsi-Hui Lin
      • Hans I Harn
      • Rei Ogawa
      • Yang-Kao Wang
      • Yen-Ting Ho
      • and others
      Cited in Scopus: 56
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        Keloids are pathological scars characterized by excessive extracellular matrix production that are prone to form in body sites with increased skin tension. CAV1, the principal coat protein of caveolae, has been associated with the regulation of cell mechanics, including cell softening and loss of stiffness sensing ability in NIH3T3 fibroblasts. Although CAV1 is present in low amounts in keloid fibroblasts (KFs), the causal association between CAV1 down-regulation and its aberrant responses to mechanical stimuli remain unclear.
        Caveolin-1 Controls Hyperresponsiveness to Mechanical Stimuli and Fibrogenesis-Associated RUNX2 Activation in Keloid Fibroblasts
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